Correction to: Mild endoplasmic reticulum stress ameliorates lipopolysaccharide-induced neuroinflammation and cognitive

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(2020) 17:353

CORRECTION

Open Access

Correction to: Mild endoplasmic reticulum stress ameliorates lipopolysaccharideinduced neuroinflammation and cognitive impairment via regulation of microglial polarization Yi-wei Wang1†, Qin Zhou1†, Xiang Zhang2†, Qing-qing Qian1, Jia-wen Xu1, Peng-fei Ni1 and Yan-ning Qian1* Correction to: J Neuroinflammation (2017) 14:233 https://doi.org/10.1186/s12974-017-1002-7 Following publication of the original article [1], the authors noticed a mistake on Fig. 3e and would like to correct it. Presented here is the corrected version of Fig. 3. Author details 1 Department of Anesthesiology, The First Affiliated Hospital of NanjingMedical University, Nanjing, Jiangsu 210029, People’s Republic of China. 2Department of Anesthesiology, Shanghai General Hospital, Shanghai JiaoTong University School of Medicine, Shanghai 200080, People’s Republic of China.

Reference 1. Wang Y, Zhou Q, Zhang X, et al. Mild endoplasmic reticulum stress ameliorates lipopolysaccharide-induced neuroinflammation and cognitive impairment via regulation of microglial polarization. J Neuroinflammation. 2017;14:233 https://doi.org/10.1186/s12974-017-1002-7.

The original article can be found online at https://doi.org/10.1186/s12974017-1002-7. * Correspondence: [email protected] † Yi-wei Wang, Qin Zhou and Xiang Zhang contributed equally to this work. 1 Department of Anesthesiology, The First Affiliated Hospital of NanjingMedical University, Nanjing, Jiangsu 210029, People’s Republic of China

© The Author(s). 2020 Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

Wang et al. Journal of Neuroinflammation

(2020) 17:353

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Fig. 3 Low doses of TM activated a nonharmful, moderate UPR in the hippocampus. The freezing time in the trace fear conditioning test (a) and the number of learning trials in the Y-maze test (b) were recorded to analyze cognitive changes (n = 12). c Immunostaining was used to detect cleaved caspase-3 in the CA1 area of the hippocampus. Scale bar, 50 μm. d Quantification of cleaved caspase-3-p