Lack of P2Y 13 in mice fed a high cholesterol diet results in decreased hepatic cholesterol content, biliary lipid secre
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Lack of P2Y13 in mice fed a high cholesterol diet results in decreased hepatic cholesterol content, biliary lipid secretion and reverse cholesterol transport Nutrition & Metabolism 2013, 10:67
doi:10.1186/1743-7075-10-67
Laeticia Lichtenstein ([email protected]) Nizar Serhan ([email protected]) Wijtske Annema ([email protected]) Guillaume Combes ([email protected]) Bernard Robaye ([email protected]) Jean-Marie Boeynaems ([email protected]) Bertrand Perret ([email protected]) Uwe J Tietge ([email protected]) Muriel Laffargue ([email protected])
ISSN Article type
1743-7075 Brief communication
Submission date
24 July 2013
Acceptance date
30 October 2013
Publication date
6 November 2013
Article URL
http://www.nutritionandmetabolism.com/content/10/1/67
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Lack of P2Y13 in mice fed a high cholesterol diet results in decreased hepatic cholesterol content, biliary lipid secretion and reverse cholesterol transport Laeticia Lichtenstein1,2,† Email: [email protected] Nizar Serhan1,2,† Email: [email protected] Wijtske Annema3 Email: [email protected] Guillaume Combes1,2 Email: [email protected] Bernard Robaye4 Email: [email protected] Jean-Marie Boeynaems4 Email: [email protected] Bertrand Perret1,5 Email: [email protected] Uwe J F Tietge3 Email: [email protected] Muriel Laffargue1,2 Email: [email protected] Laurent O Martinez1,2,5,* Email: [email protected]. 1
CHU de Toulouse, Hôpital Purpan, Toulouse, France
2
Université de Toulouse III, UMR 1048, Toulouse 31300, France
3
Department of Pediatrics, University of Groningen, University Medical Center Groningen, Groningen, Netherlands 4
Institute of Interdisciplinary Research, IRIBHM, Université Libre de Bruxelles, Gosselies, Belgium 5
INSERM U1048, Bât. L3, Hôpital Rangueil, BP 84225, 31432 Toulouse cedex 04, France
*
Corresponding author. Université de Toulouse III, UMR 1048, Toulouse 31300, France †
Equal contributors.
Abstract Background The protective effect of HDL is mostly attributed to their metabolic function in reverse cholesterol transport (RCT), a process whereby excess cellular cholesterol is taken up from peripheral cells, processed in HDL particles, and later delivered to th
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