Manual of Nonpharmacological Control of Hypertension
The prevention and correction of high blood pressure obviously is not an end in itself but a means of preventing cardiovascular complications. It is tempting to res ort to antihypertensive drugs, for the simple reason that current preparations are easy to
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Springer-Verlag Berlin Heidelberg NewYork London Paris Tokyo Hong Kong
IR. Viskoper, M.D., Assoc. Prof. The Barzilai Medical Center Ashkelon 78306 Israel
ISBN-13: 978-3-540-51070-3
e-ISBN-13: 978-3-642-74720-5
001: 10.1007/978-3-642-74720-5
Library of Congress Cataloging-in-Publication Data Manual of nonpharmacological control of hypertension / editor, J. R. Viskoper. p. CID. 1. Hypertension-Alternative treatment. 1. Viskoper, R. [DNLM: 1. Hypertension-prevention & control. WG 340 M294] RC685.H8M285 1990 616.1'3206-dc20 DNLM/DLC for Library of Congress
90-9729 CIP
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© Springer-Verlag, Berlin Heidelberg 1990 Product Liability: The publisher can give no guarantee for information about drug dosage and application thereof contained in this book. In every individual case the respective user must check its accuracy by consulting other pharmaceutical literature.
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Foreword
The prevention and correction of high blood pressure obviously is not an end in itself but a means of preventing cardiovascular complications. It is tempting to res ort to antihypertensive drugs, for the simple reason that current preparations are easy to dose and to take. There is a variety of reasons, however, to take advantage of the antihypertensive potential of nonpharmacological approaches first. For one thing, the use of tablets tends to accentuate the negative effects oflabelling a subject as hypertensive. The act oftaking tablets per se may elicit a spectrum of subjective adverse effects, as we have learned from placebo-controlled therapeutic trials. Even more importantly, the benefits of antihypertensive drug treatment are largely confined to the reduction of stroke and disease of the large vessels. With regard to incidence of cardiac events, some modest beneficial effects have been registered, such as a decrease in congestive heart failure and in the fatality rate of myocardial infarction. The effects on the incidence of coronary heart disease are only marginal at best. This disappointing finding has been ascribed to various factors: unwanted metabolie effects of antihypertensive drugs, inadvertent drops in diastolic pressure interfering with coronary perfusion, and, last but not least, failure to eliminate risk factors other than high blood pressure (smo
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