Restorative treatment in a case of amelogenesis imperfecta and 9-year follow-up: a case report
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CASE REPORT
Open Access
Restorative treatment in a case of amelogenesis imperfecta and 9-year followup: a case report Martin M. I. Sabandal1* , Till Dammaschke2 and Edgar Schäfer1
Abstract Background: Amelogenesis imperfecta is a hereditary malformation showing various manifestations regarding enamel dysplasia. This case report shows a 9-year follow-up after restorative treatment of a 16-year old female patient affected by a hypoplastic type of amelogenesis imperfecta. The caries-free, hypersensitive teeth of the patient were restored by direct dentin adhesive composite restorations performed in total etch technique. Case presentation: After rehabilitation the patient reported a marked improvement of the mastication ability and quality of life especially during food intake. Accumulation of plaque was reduced and the ability to perform adequate oral hygiene was improved. During follow-up of 9 years recurring secondary caries and debonding of fillings were recognized and retreated. Conclusions: The retrospective assessment exhibits that the performed restorative treatment prolonged the time until further treatment has to be considered, such as prosthetic treatment. Keywords: Amelogenesis imperfecta, Direct restoration, Follow-up, Restorative treatment
Background Amelogenesis imperfecta (AI) is a genetic derived development disorder of the ameloblasts during the formation of enamel. The disease is characterized by visible malformation of the enamel in a varying degree of the whole permanent and deciduous dentition, respectively [1, 2]. The prevalence of AI ranges from 1:700 to 1:14,000 [1]. Because of the various manifestations of AI Weinmann et al. suggested in 1945 a classification [3] that has been modified and adapted during the following years depending on the diagnostic possibilities. A useful classification was proposed by Witkop [2], which is based on a division of the manifestation of AI into 4 main groups with further subdivisions [2]. * Correspondence: [email protected] 1 Central Interdisciplinary Ambulance in the School of Dentistry, University of Münster, Albert-Schweitzer-Campus 1, Gebäude W30, Waldeyerstr. 30, 48149 Münster, Germany Full list of author information is available at the end of the article
Type I -hypoplastic. IA -hypoplastic, pitted autosomal dominant. IB -hypoplastic, local autosomal dominant. IC -hypoplastic, local autosomal recessive. ID -hypoplastic, smooth autosomal dominant. IE -hypoplastic, smooth X-linked dominant. IF -hypoplastic, rough autosomal dominant. IG -enamel agenesis, autosomal recessive. Type II -hypomaturation. IIA -hypomaturation, pigmented autosomal recessive. IIB -hypomaturation, X-linked recessive. IIC snowcapped teeth, X-linked. IID autosomal dominant? Type III -hypocalcified. IIIA -autosomal dominant. IIIB -autosomal recessive. Type IV -hypomaturation-hypoplastic with taurodontism. IVA -hypomaturation-hypoplastic with taurodontism, autosomal dominant.
© The Author(s). 2020 Open Access This article is licensed under a Creative Commons Attribution
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