Primary Open Angle Glaucoma
Primary open-angle glaucoma is a hereditary disease which alters the evacuation of aqueous humor, in the trabecular area. This alteration causes an increase in the intraocular pressure at pathological rates, which after some time causes anatomical damage
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 Contents 43.1
 
 The Concept of Primary Open-Angle Glaucoma ..................................................
 
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 Diagnosis: Symptomatology ....................
 
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 Current Concept of Open-Angle Glaucoma .................................................. Chamber Angle in Pure Open-Angle Glaucoma ...................................................
 
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 Goniodysgeneses or Late Congenital Glaucoma .................................................. Clinical Forms of Open-Angle Glaucoma .................................................. First Clinical Form (Scotomatous Defects) ............................... Second Clinical Form (Diffuse Defects)........................................ Third Clinical Form (Combined) ...............
 
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 Primary open-angle glaucoma is a hereditary disease which alters the evacuation of aqueous humor, in the trabecular area. This alteration causes an increase in the intraocular pressure at pathological rates, which after some time causes anatomical damage to the optic nerve and functional damage to the visual field: characteristic of glaucoma. I refer to the reader to read Chap. 2 where you will find many definitions of primary open-angle glaucoma. I repeat here the definition of Goldmann: In the Swiss textbook of Ophthalmology (1954) [1], Goldmann made the following statement:
 
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 Histology (Molecular Biology) of Trabeculectomy Specimens in a Primary Open-Angle Glaucoma (POAG) 2009 ..........................
 
 under the term Glaucoma (green cataract). Diseases are summarized, which are the consequence of rise of intraocular pressure and of which the essential is this rise of intraocular pressure.
 
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 I would also add Bruce Shields’ definition in his book, A Study Guide for Glaucoma [2]:
 
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 Cellularity and Interstitial Trabecular Tissue in Primary Open-Angle Glaucoma (POAG) ...................................
 
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 Summary...................................................
 
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 References ...............................................................
 
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 The Concept of Primary Open-Angle Glaucoma
 
 Etiopathogenesis of POAG and Goniodysgenesis ....................................... Ibopamine Test ...........................................
 
 Histology (Molecular Biology) of Trabeculectomy Specimen in Goniodysgenesis: Late Congenital Glaucoma 2009 .........................................
 
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 There is general agreement that this glaucoma, in its typical form, is defined by the following three criteria: 1.- An intraocular pressure consistently above 21 mmHg in at least one eye; 2.an open, anterior chamber angle, with no apparent ocular or systemic abnormality that might account for the elevated IOP; 3.- Typical optic nerve head damage and typical glaucomatous visual field.
 
 R. Sampaolesi et al., The Glaucomas, DOI 10.1007/978-3-642-35500-4_43, © Springer-Verlag Berlin Heidelberg 2014
 
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 43.2
 
 Diagnosis: Symptomatology
 
 The first sign is undoubtedly		
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