407 Characterization of myocardial mby CMR
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Meeting abstract
407 Characterization of myocardial mby CMR Jana Lindberg*, Kelly Han, David Lin and John R Lesser Address: Abbott Northwestern Hospital, Minneapolis, MN, USA * Corresponding author
from 11th Annual SCMR Scientific Sessions Los Angeles, CA, USA. 1–3 February 2008 Published: 22 October 2008 Journal of Cardiovascular Magnetic Resonance 2008, 10(Suppl 1):A121
doi:10.1186/1532-429X-10-S1-A121
Abstracts of the 11th Annual SCMR Scientific Sessions - 2008
Meeting abstracts – A single PDF containing all abstracts in this Supplement is available here. http://www.biomedcentral.com/content/pdf/1532-429X-10-S1-info.pdfThis abstract is available from: http://jcmr-online.com/content/10/S1/A121 © 2008 Lindberg et al; licensee BioMed Central Ltd.
Introduction Eighteen y/o female with recurrent granular cell tumor excised X3 from groin over 3 year period. After 3rd recurrence, the patient and family agreed to aggressive chemotherapy regime. An echocardiogram was performed for cardiac clearance prior to treatment which showed normal systolic LV function but a thickened lateral wall with potential mass. The diagnosis of metastatic disease, hypertrophic cardiomyopathy, primary cardiac malignancy, infiltrating disease or Fabry's disease were considered.
Purpose
tional area of mid myocardial enhancement in the mid anterior wall. The combination of 1st pass perfusion and PSIR sequences lead to a differential diagnosis of metastatic tumor or malignant or benign primary cardiac tumor. Review of the specimen from groin site excision raised suspicion for sarcoma.
Conclusion Cardiac MRI perfusion and delayed enhancement imaging is an effective noninvasive technique to characterize masses in the myocardium that may masquerade as other cardiomyopathic diseases.
Cardiac MRI was performed to characterize abnormalities seen on echocardiogram.
Methods Patient was scanned in a Siemens 1.5 T Avanto scanner. All sequences were cardiac gated. Multi plane Trufisp, HASTE, STIR, TSE T1, Fat Sat were performed. After Gadolinium administration, perfusion, Phase Sensitive Inversion recovery and TSE T1 were completed.
Results Trufisp imaging showed normal LV function with thickened basal andterolateral and inferolateral wall. On black blood TSE the thickened area is isointense and appear to be intramyocardial. Fat suppression sequences show no saturation of the thickened area. On STIR, the thickened area was hyperintense. Perfusion imaging in four chamber and short axis views showed a well defined mass measuring 2 × 3 cm. Phase Sensitive Inversion Recovery images 15 minutes post gadolinium infusion showed heterogeneous enhancement of the mass. Also noted was an addi-
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